Idiopathic pulmonary hemosiderosis as an occupational disease: a clinical case

BACKGROUND. Idiopathic pulmonary hemosiderosis (IPH) in adults is a rare pathology of unknown etiology. We did not find information about IPH with occupational hazards the available literary sources, which makes this work relevant.
 OBJECTIVE. To analyze peculiarities course patient 23 years’ experience as gas-electric welder at factories by our own clinical observation.
 RESULTS AND DISCUSSION. Changes form heterogeneous pneumatization lung parenchyma due to increased interstitial component were determined on computer tomography chest cavity (CT cavity) 1.5 years before patient’s visit, but no additional examination was prescribed, and only diagnosis “Chronic obstructive disease” (COPD) made. Negative dynamics increase miliary nodular dissemination lungs revealed CT scan after years. already detected pathohistologically videothoracoscopy right biopsy. Shortness breath cough patient, apart from treatment aimed COPD treating, other therapy prescribed (this have positive effect). The underwent comprehensive immunological examination: indicators anti-neutrophil cytoplasmic antibodies (ANCA) profile complex blood test within normal range, ruled out vasculitis an immune-mediated process. Considering Quantiferon test, examined tuberculosis excluded. Patient been suffered hemoptysis anemia moment detection first changes up present time. Due treatment, radiological stabilization process 4 months: symptoms disease decreased; small shadows areas fibrosis, are characteristic period remission, chest.
 CONCLUSIONS. features professional route are: long development manifestations (without effect therapy), absence anemia, ANCA examination, test. Corticosteroid high doses has effect, manifested mechanism may toxic (23 factories). Timely timely corticosteroid will prevent progression complications, well reduce mortality disease. Patients should be under dynamic observation evaluation CT-dynamics correction dose corticosteroids.